| Poster | Poster Session 05 Program Schedule
02/15/2024
02:30 pm - 03:45 pm
Room: Shubert Complex (Posters 1-60)
Poster Session 05: Neuropsychiatry | Addiction/Dependence | Stress/Coping | Emotional/Social Processes
Final Abstract #29
Parental Executive Functioning Abilities and Their Impact on Adolescent Impulse Regulation
Naomi Edwards, Palo Alto University, Palo Alto, United States
Robert Hickson, Palo Alto University, Palo Alto, United States
Eva Müller-Oehring, SRI International, Menlo Park, United States
Tilman Schulte, Palo Alto University, Palo Alto, United States
Category: Executive Functions/Frontal Lobes
Keyword 1: adolescence
Keyword 2: executive functions
Objective:
The objective of this study is to examine the influence of parental executive functioning abilities on adolescent impulse regulation. We hypothesized that a deficit in parental executive functioning, measured by the Barkley Deficits in Executive Functioning Scale (BDEFS), will be associated with greater adolescent impulsivity, measured by the Urgency, Premeditation, Perseverance, Sensation Seeking, and Positive Urgency Scale (UPPS-P). Our overarching aim is to contribute to the ongoing discussion surrounding strategies for bolstering the mental well-being of adolescents, particularly as it relates to impulse regulation, and how parental involvement can be a strategy to improve this.
Participants and Methods:
This study used data gathered by the ABCD Research Consortium. The study used the cross-sectional four-year follow-up data of the parent-reported BDEFS and the youth-reported UPPS-P. The sample consisted of 4,594 adolescents between the ages of 12-15. The sex assigned at birth of the sample was 2,413 male, 2,180 female, and 1 intersex. The race and ethnicity of the sample were 2,611 White, 484 Black, 936 Hispanic, 106 Asian, and 457 Other. Prior to our main analyses, we conducted a confirmatory factor analysis (CFA) in order to establish the viability of our latent measurements of adolescent impulsivity. In our primary model we regressed a latent measurement of adolescent impulsivity (observed indicators were three youth-reported UPPS-P questions) on parental executive functioning. A post hoc analysis was then conducted to see if parent gender (4090 female, 494 male, 10 other) moderated the relationship between parental executive functioning and adolescent impulsivity.
Results:
The results of the CFA for adolescent impulsivity suggested good model fit (CFI = .977, TLI = .956, SRMR = .028, RMSEA = .077, chi-square < .001) with loadings ranging from .28 to .81. The structural regression model converged with good fit (CFI = .956, TLI = .911, SRMR = .034, RMSEA = .071 chi-square < .001). The structural regression of parental executive functioning was positively associated with the latent variable of adolescent impulsivity with a moderate effect size (β = .235, p <.05). Adolescent impulsivity levels did not differ significantly based on sex assigned at birth, although sensation-seeking was higher in males than females. We did not find any significant associations using parent gender as a moderator between parental executive functioning and adolescent impulsivity.
Conclusions:
Our findings support our initial hypothesis, indicating that deficits in parental executive functioning abilities are associated with elevated levels of adolescent impulsivity. Our findings provide insight into the potential benefits of targeted parental executive functioning training as a way to improve adolescent impulsivity and offer practical implications. Further research is warranted on the longitudinal effects of inadequate parental executive functioning on the trajectory of impulse regulation in adolescents as they ascend into adulthood.
Funding: U01DA041022
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